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Editorial
. 2020 Dec 1:320:97-99.
doi: 10.1016/j.ijcard.2020.06.051. Epub 2020 Jul 4.

Mechanisms of Cav3-associated arrhythmia: Protein or microdomain dysfunction?

Antonio Zaza  1 , Eleonora Grandi  2
Affiliations
Editorial

Mechanisms of Cav3-associated arrhythmia: Protein or microdomain dysfunction?

Antonio Zaza et al. Int J Cardiol. .
No abstract available

Keywords: Caveolin-3; LQTS.

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Conflict of interest statement

CONFLICT OF INTEREST

The authors have no conflict of interest to disclose.

Figures

Figure 1:
Figure 1:. Cav-3 macromolecular complex and effects of CAV3 mutations on cardiac repolarization.
CAV3-V37L gain-of-function effects on ICaL and loss-of-function consequences on IKr and IKs are expected to prolong the cardiac action potential, as would other previously reported changes in several ion channels.
See this image and copyright information in PMC

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