Genetic Analysis of Patients With Sickle Cell Anemia and Stroke Before 4 Years of Age Suggest an Important Role for Apoliprotein E

Circ Genom Precis Med. 2020 Oct;13(5):531-540. doi: 10.1161/CIRCGEN.120.003025. Epub 2020 Sep 14.

Abstract

Background: Ischemic stroke is a devastating complication affecting children with sickle cell anemia. Genetic factors are likely to be important in determining the risk of stroke but are poorly defined.

Methods: We have studied a cohort of 19 children who had an overt ischemic stroke before 4 years of age. We predicted genetic determinants of stroke would be more prominent in this group. We performed whole exome sequencing on this cohort and applied 2 hypotheses to our variant filtering. First, we looked for strong, potentially mono- or oligogenic variants for ischemic stroke, and second, we considered that more common polygenic variants will be enriched in our cohort. Candidate variants emerging from both strategies were validated in a cohort of 283 patients with sickle cell anemia and known pediatric cerebrovascular outcomes. We used principal component analysis in this cohort to control for relatedness and population substructure.

Results: Our primary finding was that the Apoliprotein E genotypes ε2/ε4 and ε4/ ε4, defined by the interplay of rs7412 and rs429358, were associated with increased stroke risk, with an odds ratio of 4.35 ([95% CI, 1.85-10.0] P=0.0011) for ischemic stroke in the validation cohort. We also found that rs2297518 in NOS (NO synthase) 2 (odds ratio, 2.25 [95% CI, 1.21-4.19]; P=0.014) and rs2230123 in signal transducer and activator of transcription (odds ratio, 2.60 [95% CI, 1.30-5.20]; P=0.009) both had increased odds ratios for ischemic stroke, although these two variants were below the threshold for statistical significance after correction for multiple testing.

Conclusions: These data identify new loci for future functional investigations into cerebrovascular disease in sickle cell anemia. Based on African population reference allele frequencies, the Apoliprotein E genotypes would be present in about 10% of children with sickle cell anemia and represent a genetic risk factor that is potentially modifiable by both dietary and pharmaceutical manipulation of its dyslipidemic effects.

Keywords: anemia, sickle cell; gene frequency; genotype; odds ratio; risk factors.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Anemia, Sickle Cell / diagnosis
  • Anemia, Sickle Cell / genetics*
  • Apolipoproteins E / genetics*
  • CX3C Chemokine Receptor 1 / genetics
  • Child, Preschool
  • Collagen Type VII / genetics
  • Gene Frequency
  • Genotype
  • Humans
  • Membrane Proteins / genetics
  • Multifactorial Inheritance / genetics
  • Nerve Tissue Proteins / genetics
  • Odds Ratio
  • Polymorphism, Single Nucleotide
  • Protein Subunits / genetics
  • Risk Factors
  • Stroke / diagnosis
  • Stroke / genetics*

Substances

  • Apolipoproteins E
  • COL7A1 protein, human
  • CX3C Chemokine Receptor 1
  • CX3CR1 protein, human
  • Collagen Type VII
  • Membrane Proteins
  • Nerve Tissue Proteins
  • Protein Subunits
  • TENM3 protein, human