Does heterozygosity for UGT1A1 *28 convey increased risk for severe neonatal jaundice?
J Perinatol
.
2021 Apr;41(4):658-660.
doi: 10.1038/s41372-020-00826-5.
Epub 2020 Sep 21.
Authors
Timothy M Bahr
1
,
Archana M Agarwal
2
,
Robert D Christensen
3
4
Affiliations
1
Division of Neonatology, Department of Pediatrics, University of Utah Health, Salt Lake City, UT, USA. Tim.bahr@hsc.utah.edu.
2
Division of Hematopathology, Department of Pathology, University of Utah Health and ARUP Laboratories, Salt Lake City, UT, USA.
3
Division of Neonatology, Department of Pediatrics, University of Utah Health, Salt Lake City, UT, USA.
4
Division of Hematology/Oncology, Department of Pediatrics, University of Utah Health, Salt Lake City, UT, USA.
PMID:
32958836
DOI:
10.1038/s41372-020-00826-5
No abstract available
MeSH terms
Glucuronosyltransferase / genetics*
Glucuronosyltransferase / metabolism
Humans
Hyperbilirubinemia, Neonatal*
Infant, Newborn
Jaundice, Neonatal* / genetics
Substances
UGT1A1 enzyme
Glucuronosyltransferase