Ketogenic diet feeding improves aerobic metabolism property in extensor digitorum longus muscle of sedentary male rats

PLoS One. 2020 Oct 30;15(10):e0241382. doi: 10.1371/journal.pone.0241382. eCollection 2020.

Abstract

Recent studies of the ketogenic diet, an extremely high-fat diet with extremely low carbohydrates, suggest that it changes the energy metabolism properties of skeletal muscle. However, ketogenic diet effects on muscle metabolic characteristics are diverse and sometimes countervailing. Furthermore, ketogenic diet effects on skeletal muscle performance are unknown. After male Wistar rats (8 weeks of age) were assigned randomly to a control group (CON) and a ketogenic diet group (KD), they were fed for 4 weeks respectively with a control diet (10% fat, 10% protein, 80% carbohydrate) and a ketogenic diet (90% fat, 10% protein, 0% carbohydrate). After the 4-week feeding period, the extensor digitorum longus (EDL) muscle was evaluated ex vivo for twitch force, tetanic force, and fatigue. We also analyzed the myosin heavy chain composition, protein expression of metabolic enzymes and regulatory factors, and citrate synthase activity. No significant difference was found between CON and KD in twitch or tetanic forces or muscle fatigue. However, the KD citrate synthase activity and the protein expression of Sema3A, citrate synthase, succinate dehydrogenase, cytochrome c oxidase subunit 4, and 3-hydroxyacyl-CoA dehydrogenase were significantly higher than those of CON. Moreover, a myosin heavy chain shift occurred from type IIb to IIx in KD. These results demonstrated that the 4-week ketogenic diet improves skeletal muscle aerobic capacity without obstructing muscle contractile function in sedentary male rats and suggest involvement of Sema3A in the myosin heavy chain shift of EDL muscle.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Diet, Ketogenic*
  • Energy Metabolism*
  • Glycogen / metabolism
  • Male
  • Muscle Contraction
  • Muscle Fatigue
  • Muscle, Skeletal / physiology*
  • Rats, Wistar
  • Sedentary Behavior

Substances

  • Glycogen

Grants and funding

This study was supported by a Grant-in-Aid for Exploratory Research (24650334 to CK) and by a Grant-in-Aid for Scientific Research (18K17767 to CK) from the Ministry of Education, Culture, Sports, Science, and Technology of Japan (MEXT).