Reduced excretion of vasodilator prostaglandins in preeclampsia

Agents Actions Suppl. 1987:22:175-81. doi: 10.1007/978-3-0348-9299-5_18.

Abstract

The role of prostaglandin (PG) system in preeclampsia (pre-E) was investigated. Urinary excretion of PGE2,6-keto PGF1 alpha,2,3 dinor 6-keto PGF1 alpha, TxB2 and 2,3-dinor-TxB2 and kallikrein were determined in 10 normotensive pregnant women and 14 with pre-E. 6-keto PGF1 alpha and 2,3-dinor 6-keto PGF1 alpha (the main renal and extrarenal metabolites of vasodilator PGI2) and PGE2 excretion was lower in pre-E. TxB2 metabolites in urine were similar in the two groups of women. Our data are consistent with the hypothesis of an imbalance between vasodilator and vasoconstrictor PGs in pre-E.

MeSH terms

  • 6-Ketoprostaglandin F1 alpha / analogs & derivatives
  • 6-Ketoprostaglandin F1 alpha / urine
  • Adult
  • Dinoprostone
  • Epoprostenol / metabolism
  • Female
  • Humans
  • Kidney / metabolism
  • Pre-Eclampsia / metabolism
  • Pre-Eclampsia / urine*
  • Pregnancy
  • Prostaglandins / urine*
  • Prostaglandins E / urine
  • Thromboxane A2 / metabolism
  • Thromboxane B2 / urine
  • Vasodilator Agents / urine*

Substances

  • Prostaglandins
  • Prostaglandins E
  • Vasodilator Agents
  • Thromboxane B2
  • Thromboxane A2
  • 6-Ketoprostaglandin F1 alpha
  • 2,3-dinor-6-ketoprostaglandin F1alpha
  • Epoprostenol
  • Dinoprostone