Effects of ethanol on neurotransmitter release and intracellular free calcium in PC12 cells

J Pharmacol Exp Ther. 1988 Feb;244(2):417-22.

Abstract

The effect of ethanol on muscarine-stimulated release of l-[3H]norepinephrine ([3H]NE) was studied using the rat pheochromocytoma cell line, PC12. At concentrations of 25 mM and above, ethanol produced a dose-dependent inhibition of muscarine-stimulated release of [3H]NE. The inhibition of muscarine-stimulated transmitter release occurred in the absence of any detectable effect of ethanol on [3H]NE uptake or on muscarinic binding to the cells. However, ethanol produced an inhibition of muscarine-stimulated elevation of intracellular free Ca++ which corresponded with the inhibition of transmitter release. At concentrations greater than 100 mM, ethanol produced an increase in the basal release of [3H]NE. Intracellular free Ca++ also was increased by ethanol concentrations greater than 100 mM. The elevation of basal transmitter release and intracellular free Ca++ by concentrations of ethanol greater than 100 mM occurred independently of the inhibition by ethanol of muscarine-stimulated elevation of intracellular free Ca++ and transmitter secretion. These results suggest that the effects of ethanol on neurotransmitter release are associated with the effects of ethanol on intracellular free Ca++.

MeSH terms

  • Adrenal Gland Neoplasms / metabolism
  • Animals
  • Calcium / analysis*
  • Ethanol / pharmacology*
  • Muscarine / pharmacology
  • N-Methylscopolamine
  • Norepinephrine / metabolism*
  • Pheochromocytoma / metabolism
  • Rats
  • Receptors, Muscarinic / metabolism
  • Scopolamine Derivatives / metabolism
  • Tritium
  • Tumor Cells, Cultured

Substances

  • Receptors, Muscarinic
  • Scopolamine Derivatives
  • Tritium
  • Ethanol
  • Muscarine
  • Calcium
  • N-Methylscopolamine
  • Norepinephrine