Low-intensity exercise diverts cardiac fatty acid metabolism from triacylglycerol synthesis to beta oxidation in fructose-fed rats

Arch Physiol Biochem. 2023 Dec;129(4):922-932. doi: 10.1080/13813455.2021.1886118. Epub 2021 Feb 21.

Abstract

Context: Excessive fructose consumption causes ectopic lipid storage leading to metabolic disorders and cardiovascular diseases associated with defective substrate utilisation in the heart.

Objective: Examining the preventive impact of low-intensity exercise on alterations related to fructose-rich diet (FRD) on cardiac fatty acid (FA) transport and metabolism.

Materials and methods: Male Wistar rats were divided into control and two groups that received 10% fructose for 9 weeks, one of which was additionally exposed to exercise.

Results: FRD elevated plasma and cardiac TAG, FATP1 in plasma membrane, Lipin 1 in microsomes and HSL mRNA, while mitochondrial CPT1 was decreased. Exercise decreased plasma free FA level, raised CD36 in plasma membrane and FATP1 in lysate, mitochondrial CPT1 and decreased microsomal Lipin 1 in fructose-fed rats.

Conclusions: FRD changed plasma lipids and augmented partitioning of FA to TAG storage in the heart, whereas exercise in FRD rats switched metabolism of FA towards β-oxidation.

Keywords: Exercise; fatty acid metabolism; fatty acid transport; fructose-rich diet; heart.

MeSH terms

  • Animals
  • Fatty Acids / metabolism
  • Fructose*
  • Lipid Metabolism*
  • Male
  • Organic Chemicals
  • Rats
  • Rats, Wistar
  • Triglycerides / metabolism

Substances

  • Fructose
  • Fatty Acids
  • Triglycerides
  • lipine
  • Organic Chemicals