Transcriptome-wide association study for postpartum depression implicates altered B-cell activation and insulin resistance

Mol Psychiatry. 2022 Jun;27(6):2858-2867. doi: 10.1038/s41380-022-01525-7. Epub 2022 Apr 1.

Abstract

Postpartum depression (PPD) affects 1 in 7 women and has negative mental health consequences for both mother and child. However, the precise biological mechanisms behind the disorder are unknown. Therefore, we performed the largest transcriptome-wide association study (TWAS) for PPD (482 cases, 859 controls) to date using RNA-sequencing in whole blood and deconvoluted cell types. No transcriptional changes were observed in whole blood. B-cells showed a majority of transcriptome-wide significant results (891 transcripts representing 789 genes) with pathway analyses implicating altered B-cell activation and insulin resistance. Integration of other data types revealed cell type-specific DNA methylation loci and disease-associated eQTLs (deQTLs), but not hormones/neuropeptides (estradiol, progesterone, oxytocin, BDNF), serve as regulators for part of the transcriptional differences between cases and controls. Further, deQTLs were enriched for several brain region-specific eQTLs, but no overlap with MDD risk loci was observed. Altogether, our results constitute a convergence of evidence for pathways most affected in PPD with data across different biological mechanisms.

MeSH terms

  • Depression, Postpartum* / genetics
  • Depression, Postpartum* / metabolism
  • Female
  • Genome-Wide Association Study* / methods
  • Humans
  • Insulin Resistance* / genetics
  • Transcriptome / genetics