Comprehensive Comparison of Novel Bovine Leukemia Virus (BLV) Integration Sites between B-Cell Lymphoma Lines BLSC-KU1 and BLSC-KU17 Using the Viral DNA Capture High-Throughput Sequencing Method

Viruses. 2022 May 7;14(5):995. doi: 10.3390/v14050995.

Abstract

Bovine leukemia virus (BLV) infects cattle and integrates into host DNA, causing enzootic bovine leukosis (EBL), an aggressive B-cell lymphoma. Here, we developed a novel proviral DNA-capture sequencing (proviral DNA-capture-seq) method investigating BLV proviral integration in two B-cell lymphoma lines, BLSC-KU1 and BLSC-KU17, derived from BLV-infected cattle with EBL. We designed BLV-specific biotinylated probes to capture the provirus genome and enrich libraries for next-generation sequencing. Validation showed high specificity and efficient enrichment of target sequence reads as well as identification of three BLV proviral integration sites on BLV persistently infected FLK-BLV cells as a positive control. We successfully detected a single BLV proviral integration site on chromosome 19 of BLSC-KU1 and chromosome 9 of BLSC-KU17, which were confirmed by standard PCR and Sanger sequencing. Further, a defective provirus in BLSC-KU1 and complete BLV proviral sequence in BLSC-KU17 were confirmed using long PCR and sequencing. This is the first study to provide comprehensive information on BLV proviral structure and viral integration in BLSC-KU1 and BLSC-KU17. Moreover, the proposed method can facilitate understanding of the detailed mechanisms underlying BLV-induced leukemogenesis and may be used as an innovative tool to screen BLV-infected cattle at risk at an earlier stage than those that have already developed lymphoma.

Keywords: B-cell lymphoma line; PCR; bovine leukemia virus (BLV); integration site; next-generation sequencing (NGS); proviral DNA capture sequencing.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cattle
  • DNA, Viral / genetics
  • Enzootic Bovine Leukosis*
  • High-Throughput Nucleotide Sequencing
  • Leukemia Virus, Bovine* / genetics
  • Lymphoma, B-Cell* / genetics
  • Lymphoma, B-Cell* / veterinary
  • Proviruses / genetics

Substances

  • DNA, Viral

Grants and funding

This work was funded by a Grant-in-Aid for Scientific Research (A) from the Japan Society for the Promotion of Science (JSPS) (grant number 16H02590), the JSPS Postdoctoral Fellowship (grant number 18F18100), and grants from Livestock Promotional Subsidy from the Japan Racing Association.