Restless legs syndrome, neuroleptic-induced akathisia, and opioid-withdrawal restlessness: shared neuronal mechanisms?

Sleep. 2024 Mar 11;47(3):zsad273. doi: 10.1093/sleep/zsad273.

Abstract

Restlessness is a core symptom underlying restless legs syndrome (RLS), neuroleptic-induced akathisia, and opioid withdrawal. These three conditions also share other clinical components suggesting some overlap in their pathophysiology. Recent prospective studies demonstrate the frequent incidence of RLS-like symptoms during opioid withdrawal and supervised prescription opioid tapering. Based on the therapeutic role of µ-opioid receptor (MOR) agonists in the three clinical conditions and recent preclinical experimental data in rodents, we provide a coherent and unifying neurobiological basis for the restlessness observed in these three clinical syndromes and propose a heuristic hypothesis of a key role of the specific striatal neurons that express MORs in akathisia/restlessness.

Keywords: akathisia; neuroleptics; opioid withdrawal; restless legs syndrome.

MeSH terms

  • Analgesics, Opioid / adverse effects
  • Antipsychotic Agents* / therapeutic use
  • Humans
  • Psychomotor Agitation / etiology
  • Restless Legs Syndrome* / diagnosis

Substances

  • Analgesics, Opioid
  • Antipsychotic Agents