The Switch/Sucrose Nonfermentable Subunit ARID1A Mediates Neutrophil-Associated Skin Inflammatory Responses

J Invest Dermatol. 2024 Oct;144(10):2324-2328.e4. doi: 10.1016/j.jid.2024.03.015. Epub 2024 Mar 28.

¹ Department of Dermatology, University of California San Diego, San Diego, California, USA; Department of Cellular and Molecular Medicine, University of California San Diego, San Diego, California, USA; Stem Cell Program, University of California San Diego, San Diego, California, USA.
² Department of Dermatology, University of California San Diego, San Diego, California, USA; Department of Cellular and Molecular Medicine, University of California San Diego, San Diego, California, USA; Stem Cell Program, University of California San Diego, San Diego, California, USA; Department of Dermatology, Wonju College of Medicine, Yonsei University, Wonju, Republic of Korea.
³ Department of Dermatology, University of California San Diego, San Diego, California, USA; Department of Cellular and Molecular Medicine, University of California San Diego, San Diego, California, USA; Stem Cell Program, University of California San Diego, San Diego, California, USA. Electronic address: gsen@ucsd.edu.

No abstract available

Keywords: ARID1A; Cytokines; Innate immune response; SWI/SNF; Skin inflammation.

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