Lipopolysaccharide and lipid A-induced human blood lymphocyte activation as detected by a protein A plaque assay

Eur J Immunol. 1979 Aug;9(8):619-25. doi: 10.1002/eji.1830090809.

Abstract

Various purified cell wall lipopolysaccharides (LPS) from gram-negative bacteria and derivatives of these LPS were tested for their stimulatory capacity for human peripheral blood cells. Immunoglobulin (Ig) production was tested by an indirect plaque-forming cell assay using Staphylococcus aureus protein A-coupled erythrocytes and specific anti-Ig as developing serum. This method allows the detection of the majority of cells secreting Ig of a single class, and the number of plaque-forming cells detected are approximately 100-1000 times the amount obtained using normal sheep red cells as targets. LPS containing the O antigen-specific chain, as well as mutant products only containing lipid A and ketodeoxyoctonate trisaccharide, could induce cell division and antibody synthesis. The polypeptide antibiotic polymyxin B was found to inhibit LPS-induced activation. Furthermore, purified lipid A, complexed with bovine serum albumin, was also found to activate human peripheral blood B cells. These findings demonstrate that human peripheral blood lymphocytes can be activated by LPS and also indicate that lipid A is the active part of these molecules.

MeSH terms

  • DNA / biosynthesis
  • Dose-Response Relationship, Immunologic
  • Hemolytic Plaque Technique*
  • Humans
  • Immunoglobulin A / biosynthesis
  • Immunoglobulin G / biosynthesis
  • Immunoglobulin M / biosynthesis
  • Lipid A / pharmacology*
  • Lipopolysaccharides / pharmacology*
  • Lymphocyte Activation*
  • Staphylococcal Protein A / pharmacology*

Substances

  • Immunoglobulin A
  • Immunoglobulin G
  • Immunoglobulin M
  • Lipid A
  • Lipopolysaccharides
  • Staphylococcal Protein A
  • DNA