Enrichment for the POLE mutated against p53 wild subtype using clinicopathologic factors and cyclin B1 immunohistochemistry in endometrial cancer

J Gynecol Oncol. 2024 May;35(3):e94. doi: 10.3802/jgo.2024.35.e94.
No abstract available

Publication types

  • Case Reports
  • Letter

MeSH terms

  • Adult
  • Aged
  • Cyclin B1* / genetics
  • Cyclin B1* / metabolism
  • DNA Polymerase II* / genetics
  • DNA Polymerase II* / metabolism
  • Endometrial Neoplasms* / genetics
  • Endometrial Neoplasms* / pathology
  • Female
  • Humans
  • Immunohistochemistry*
  • Middle Aged
  • Mutation
  • Poly-ADP-Ribose Binding Proteins* / genetics
  • Tumor Suppressor Protein p53* / genetics
  • Tumor Suppressor Protein p53* / metabolism

Substances

  • POLE protein, human
  • TP53 protein, human
  • CCNB1 protein, human