The Adaptive Immune Response in Hepatitis B Virus-Associated Hepatocellular Carcinoma Is Characterized by Dysfunctional and Exhausted HBV-Specific T Cells

Viruses. 2024 Apr 29;16(5):707. doi: 10.3390/v16050707.

Abstract

This systematic review investigates the immunosuppressive environment in HBV-associated hepatocellular carcinoma (HCC), characterized by dysfunctional and exhausted HBV-specific T cells alongside an increased infiltration of HBV-specific CD4+ T cells, particularly regulatory T cells (Tregs). Heightened expression of checkpoint inhibitors, notably PD-1, is linked with disease progression and recurrence, indicating its potential as both a prognostic indicator and a target for immunotherapy. Nevertheless, using PD-1 inhibitors has shown limited effectiveness. In a future perspective, understanding the intricate interplay between innate and adaptive immune responses holds promise for pinpointing predictive biomarkers and crafting novel treatment approaches for HBV-associated HCC.

Keywords: HBV-specific T-cell response; hepatitis B virus; hepatocellular carcinoma; immune response.

Publication types

  • Systematic Review
  • Review

MeSH terms

  • Adaptive Immunity*
  • CD4-Positive T-Lymphocytes / immunology
  • Carcinoma, Hepatocellular* / immunology
  • Carcinoma, Hepatocellular* / virology
  • Hepatitis B / complications
  • Hepatitis B / immunology
  • Hepatitis B / virology
  • Hepatitis B virus* / immunology
  • Hepatitis B, Chronic / complications
  • Hepatitis B, Chronic / immunology
  • Hepatitis B, Chronic / virology
  • Humans
  • Immune Checkpoint Inhibitors / pharmacology
  • Immune Checkpoint Inhibitors / therapeutic use
  • Immunotherapy
  • Liver Neoplasms* / immunology
  • Liver Neoplasms* / virology
  • Programmed Cell Death 1 Receptor / immunology
  • T-Lymphocytes / immunology
  • T-Lymphocytes, Regulatory / immunology

Substances

  • Programmed Cell Death 1 Receptor
  • Immune Checkpoint Inhibitors

Grants and funding

This research received no external funding.