Obox4 promotes zygotic genome activation upon loss of Dux

Elife. 2024 Jun 24:13:e95856. doi: 10.7554/eLife.95856.

Abstract

Once fertilized, mouse zygotes rapidly proceed to zygotic genome activation (ZGA), during which long terminal repeats (LTRs) of murine endogenous retroviruses with leucine tRNA primer (MERVL) are activated by a conserved homeodomain-containing transcription factor, DUX. However, Dux-knockout embryos produce fertile mice, suggesting that ZGA is redundantly driven by an unknown factor(s). Here, we present multiple lines of evidence that the multicopy homeobox gene, Obox4, encodes a transcription factor that is highly expressed in mouse two-cell embryos and redundantly drives ZGA. Genome-wide profiling revealed that OBOX4 specifically binds and activates MERVL LTRs as well as a subset of murine endogenous retroviruses with lysine tRNA primer (MERVK) LTRs. Depletion of Obox4 is tolerated by embryogenesis, whereas concomitant Obox4/Dux depletion markedly compromises embryonic development. Our study identified OBOX4 as a transcription factor that provides genetic redundancy to preimplantation development.

Keywords: developmental biology; homeobox; mouse; preimplantation development; retrotransposon; transcription factors; zygotic genome activation.

MeSH terms

  • Animals
  • Embryonic Development / genetics
  • Gene Expression Regulation, Developmental
  • Genome
  • Homeodomain Proteins* / genetics
  • Homeodomain Proteins* / metabolism
  • Mice
  • Mice, Knockout
  • Zygote* / metabolism

Substances

  • Homeodomain Proteins
  • Dux4 protein, mouse

Associated data

  • GEO/GSE196671
  • GEO/GSE45719
  • GEO/GSE165782
  • GEO/GSE168728
  • GEO/GSE121746
  • GEO/GSE141321
  • GEO/GSE85632