Selenomethionine Inhibited HADV-Induced Apoptosis Mediated by ROS through the JAK-STAT3 Signaling Pathway

Nutrients. 2024 Jun 20;16(12):1966. doi: 10.3390/nu16121966.

Abstract

Adenovirus (HAdV) can cause severe respiratory infections in children and immunocompromised patients. There is a lack of specific therapeutic drugs for HAdV infection, and the study of anti-adenoviral drugs has far-reaching clinical implications. Elemental selenium can play a specific role as an antioxidant in the human immune cycle by non-specifically binding to the amino acid methionine in body proteins. Methods: The antiviral mechanism of selenomethionine was explored by measuring cell membrane status, intracellular DNA status, cytokine secretion, mitochondrial membrane potential, and ROS production. Conclusions: Selenomethionine improved the regulation of ROS-mediated apoptosis by modulating the expression of Jak1/2, STAT3, and BCL-XL, which led to the inhibition of apoptosis. It is anticipated that selenomethionine will offer a new anti-adenoviral therapeutic alternative.

Keywords: ROS; adenovirus; apoptosis; selenomethionine.

MeSH terms

  • A549 Cells
  • Antiviral Agents / pharmacology
  • Apoptosis* / drug effects
  • Humans
  • Janus Kinases / metabolism
  • Membrane Potential, Mitochondrial / drug effects
  • Reactive Oxygen Species* / metabolism
  • STAT3 Transcription Factor / metabolism
  • Selenomethionine* / pharmacology
  • Signal Transduction* / drug effects

Substances

  • Antiviral Agents
  • Janus Kinases
  • Reactive Oxygen Species
  • Selenomethionine
  • STAT3 protein, human
  • STAT3 Transcription Factor