Abstract
The role of type I interferon (IFN-I) in systemic lupus erythematosus (SLE) is well documented, but the role of interleukin (IL)-1β remains elusive. In this issue of Immunity, Caielli et al. identified an SLE monocyte population coproducing IL-1β and IFN-I and described how mitochondrial nucleic-acid-containing RBCs engage cGAS/STING, RIG-I, MDA5, and NLRP3 for unconventional IL-1β release.
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Publication types
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Research Support, N.I.H., Extramural
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Comment
MeSH terms
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Animals
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Humans
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Interferon Type I / immunology
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Interferon Type I / metabolism
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Interleukin-1beta* / immunology
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Interleukin-1beta* / metabolism
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Lupus Erythematosus, Systemic* / immunology
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Lupus Erythematosus, Systemic* / metabolism
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Mitochondria* / immunology
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Mitochondria* / metabolism
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Monocytes* / immunology
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Monocytes* / metabolism
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Myxovirus Resistance Proteins / genetics
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Myxovirus Resistance Proteins / metabolism
Substances
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Interleukin-1beta
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Interferon Type I
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Myxovirus Resistance Proteins