Ruminative thinking mediates the effects of exposure to adverse life events on psychotic-like experiences

Front Psychol. 2024 Nov 12:15:1434470. doi: 10.3389/fpsyg.2024.1434470. eCollection 2024.

Abstract

Introduction: A growing literature has shown that exposure to adverse life events during childhood or adolescence is associated with the presence of psychotic-like experiences (PLEs), which is in turn associated with the risk of psychotic outcomes. Ruminative thinking, i.e., the tendency to dwell on particular issues or ideas, may affect the perceived aversiveness and ability to cope with adverse life events. However, the role that rumination plays in the relationship between adverse life events and the presence of PLEs remains unclear. The purpose of this study is to assess the association between adverse life events and PLEs in a longitudinal sample of young adults and adolescents, and to investigate whether this relationship is mediated by ruminative thinking.

Methods: We used a longitudinal naturalistic sample of 706 volunteers assessed at ages 18 and 22 years, within the Imagen consortium. Lifetime occurrence of adverse life events (i.e., events perceived as strongly negative by participants) was investigated using the Life Events Questionnaire. The Community Assessment of Psychic Experience (CAPE-42) served to assess the presence of PLEs, while ruminative thinking was investigated through the Ruminative Response Scale.

Results: Results showed that both frequency of PLEs and their persistence over time were associated with greater adverse life events exposure (r = 0.32, p < 0.001 and F 1 = 9.8; p < 0.001, respectively) and greater ruminative response (r = 0.66, p < 0.001 and F 1 = 94.9; p < 0.001, respectively). Mediation analyses showed that relationship between adverse life events and PLEs frequency was partially mediated by rumination (direct effect Z: 5.4, p < 0.001; indirect effect Z: 6.9, p < 0.001; total effect Z: 5.9, p < 0.001). Considering changes between the two assessment timepoints, relationship between PLEs variation between 18 and 22 years and adverse life events occurred during the same period was partially mediated by changes in rumination (direct effect Z: 2.8, p < 0.005; indirect effect Z: 4.3, p < 0.001; total effect Z: 4.3; p < 0.001).

Discussion: Overall, our findings confirm that the presence of adverse life events may increase the risk of experiencing PLEs in healthy individuals and suggest that dysfunctional coping strategies, such as ruminative thinking, may be related to psychosis proneness. Results do not disentangle whether individuals with greater risk for psychosis tend to ruminate more or whether rumination exacerbates psychosis risk.

Keywords: adverse life events; clinical psychology; psychosis risk; psychotic-like experiences; rumination; ruminative response.

Grants and funding

The author(s) declare financial support was received for the research, authorship, and/or publication of this article. This work received funding from the European Union through the European Union-funded FP6 Integrated Project IMAGEN (Reinforcement-related behavior in normal brain function and psychopathology; LSHM-CT- 2007-037286), the Horizon 2020 funded ERC Advanced Grant: STRATIFY (Brain network based stratification of reinforcement-related disorders) (695313), Horizon Europe: environMENTAL grant no: 101057429, UK Research and Innovation (UKRI) Horizon Europe funding guarantee (10041392 and 10038599), Human Brain Project (HBP SGA 2 785907 and HBP SGA 3 945539), and the Chinese government via the Ministry of Science and Technology (MOST). The German Center for Mental Health (DZPG) the Bundesministerium für Bildung und Forschung (BMBF grants 01GS08152, 01EV0711, Forschungsnetz AERIAL 01EE1406A 01EE1406B, and Forschungsnetz IMAC-Mind 01GL1745B), the Deutsche Forschungsgemeinschaft (DFG grants186318919 [FOR 1617] 178833530 [SFB 940] 386691645 [NE 1383/14-1] 402170461 [TRR 265] 454245598 [IRTG 2773]), the Medical Research Foundation and Medical Research Council (grants MR/R00465X/1 and MR/S020306/1), the National Institutes of Health (NIH) funded ENIGMA-grants 5U54EB020403-05 1R56AG058854-01 and U54 EB020403 as well as NIH R01DA049238 the National Institutes of Health Science Foundation Ireland (16/ERCD/3797). NSFC grant 82150710554. Further support was provided by grants from the ANR (ANR-12-SAMA-0004 AAPG2019—GeBra), the Eranet Neuron (AF12-NEUR0008-01—WM2NA and ANR-18-NEUR00002-01—ADORe), the Fondation de France (00081242), the Fondation pour la Recherche Médicale (DPA20140629802), the Mission Interministérielle de Lutte-contre-les-Drogues-et-les-Conduites-Addictives (MILDECA), the Assistance-Publique-Hôpitaux-de-Paris and INSERM (interface grant) Paris Sud University IDEX 2012 the Fondation de l'Avenir (grant AP-RM-17-013), and the Fédération pour la Recherche sur le Cerveau. This project has received funding from the Italian Ministry for Universities and Research (MUR)—Research Projects of National Relevance (PRIN) 2022, CUP H53D23004250006 awarded to LF and GP; from The Italian Ministry for Universities and Research (MUR)—Research Projects of National Relevance (PRIN) 2020, CUP H95F21001830001, awarded to GP, and from Complementary National Plan PNC-I.1: Research initiatives for innovative technologies and pathways in the health and welfare sector, D.D. 931 of 06/06/2022, DARE—DigitAl lifelong pRevEntion initiative, code PNC0000002, CUP B53C22006420001, awarded to LA and AB.