Exploratory biomarker analysis from a phase III study of the PI3K inhibitor, copanlisib, in combination with rituximab in patients with indolent non-Hodgkin lymphoma, a retrospective study

Clin Transl Oncol. 2025 Aug;27(8):3439-3448. doi: 10.1007/s12094-025-03869-2. Epub 2025 Feb 21.

Abstract

Purpose: There has been increased difficulty in developing safe and effective treatment using PI3K inhibitors in heme malignancies, despite the role of PI3K/AKT being well defined in this population. This study was an attempt to conduct exploratory biomarker analysis retrospectively from the phase III CHRONOS-3 trial with the aim to identify a sub-set of patients that could benefit from treatment.

Patients and methods: Patients with CD20-positive indolent B-cell lymphoma were randomized 2:1 to receive intravenous copanlisib plus rituximab (C + R) or placebo plus rituximab (P + R). Biomarker analyses were performed to examine potential associations between treatment outcome and phosphatase and tensin homolog (PTEN) protein expression, EZH2 and BCL2 mutation status via next-generation sequencing, and plasma cytokine levels.

Results: PTEN presence was associated with significant improvements in progression-free survival (PFS) for C + R over P + R in patients with iNHL (P = 0.001) and FL (P = 0.012). Both the mutant and wild-type EZH2 FL patients had equal PFS benefits when treated with copanlisib. A significant improvement in PFS was observed for patients with mutant versus wild-type BCL2 FL in the C + R arm (P = 0.002). Overall survival (OS) was significantly improved for patients with iNHL and low or undetectable versus high baseline IL-2 levels in the C + R arm (P < 0.0001, unadjusted).

Conclusions: PTEN presence, BCL2 mutations, and low or undetectable baseline IL-2 levels were associated with improved patient survival following treatment with C + R, supporting a potential role for these biomarkers in guiding treatment selection for patients with indolent non-Hodgkin lymphoma.

Keywords: Biomarkers; Copanlisib; Indolent non-Hodgkin lymphoma; Phase III clinical trials; Rituximab.

Publication types

  • Clinical Trial, Phase III
  • Randomized Controlled Trial

MeSH terms

  • Adult
  • Aged
  • Antineoplastic Combined Chemotherapy Protocols* / therapeutic use
  • Biomarkers, Tumor* / genetics
  • Biomarkers, Tumor* / metabolism
  • Enhancer of Zeste Homolog 2 Protein / genetics
  • Female
  • Humans
  • Lymphoma, Non-Hodgkin* / drug therapy
  • Lymphoma, Non-Hodgkin* / genetics
  • Male
  • Middle Aged
  • Mutation
  • PTEN Phosphohydrolase / genetics
  • PTEN Phosphohydrolase / metabolism
  • Phosphoinositide-3 Kinase Inhibitors* / therapeutic use
  • Progression-Free Survival
  • Proto-Oncogene Proteins c-bcl-2 / genetics
  • Pyrimidines / administration & dosage
  • Quinazolines / administration & dosage
  • Retrospective Studies
  • Rituximab / administration & dosage

Substances

  • Rituximab
  • copanlisib
  • Biomarkers, Tumor
  • PTEN Phosphohydrolase
  • PTEN protein, human
  • Proto-Oncogene Proteins c-bcl-2
  • Pyrimidines
  • Quinazolines
  • Phosphoinositide-3 Kinase Inhibitors
  • Enhancer of Zeste Homolog 2 Protein
  • BCL2 protein, human
  • EZH2 protein, human