Inhibition of miR-325 inhibits KIF20B expression and the colorectal cancer cells' invasion & proliferation

BMC Cancer. 2025 Apr 14;25(1):680. doi: 10.1186/s12885-025-13759-z.

Abstract

Objective: This study aimed to investigate the effect of miR-325 on KIF20B expression and its role in the invasion and proliferation of colorectal cancer cells.

Methods: Colorectal cancer HCT116 cells were cultured and transfected with a miR-325 inhibitor. KIF20B expression was assessed using quantitative polymerase chain reaction (qPCR) and western blotting. Cell proliferation was assessed with the Cell Counting Kit-8 (CCK8) assay, while invasion was evaluated using Transwell and scratch wound healing assays. The expression levels of the invasion-related proteins Matrix Metalloproteinase-2 (MMP-2) and MMP-9 were also analyzed.

Results: The q-PCR and western blot results demonstrated that KIF20B expression was significantly reduced by miR-325 inhibition. The CCK8 assay revealed that miR-325 inhibition decreased cell proliferation. Furthermore, Transwell and Scratch Wound Healing assays showed that miR-325 inhibition suppressed the invasive capacity of colorectal cancer cells. The inhibition of miR-325 also led to decreased expression levels of MMP-2 and MMP-9.

Conclusion: miR-325 inhibition effectively suppresses KIF20B expression, reducing the invasion and proliferation of colorectal cancer cells, suggesting miR-325 as a potential therapeutic target.

Keywords: Colorectal cancer; Invasion; KIF20B; Proliferation.

MeSH terms

  • Cell Line, Tumor
  • Cell Movement / genetics
  • Cell Proliferation / genetics
  • Colorectal Neoplasms* / genetics
  • Colorectal Neoplasms* / metabolism
  • Colorectal Neoplasms* / pathology
  • Gene Expression Regulation, Neoplastic*
  • HCT116 Cells
  • Humans
  • Kinesins* / genetics
  • Kinesins* / metabolism
  • Matrix Metalloproteinase 2 / genetics
  • Matrix Metalloproteinase 2 / metabolism
  • Matrix Metalloproteinase 9 / genetics
  • Matrix Metalloproteinase 9 / metabolism
  • MicroRNAs* / antagonists & inhibitors
  • MicroRNAs* / genetics
  • Neoplasm Invasiveness / genetics

Substances

  • MicroRNAs
  • Kinesins
  • Matrix Metalloproteinase 2
  • Matrix Metalloproteinase 9
  • MMP2 protein, human