Abstract
Bone marrow CD8+ T cells from patients with MDS and CMML show decreased TCR diversity compared to AML. Treatment with the hypomethylating agent azacitidine in MDS and CMML alters TCR repertoire of CD8+ T cells without affecting TCR diversity.
Keywords:
CD8+ T cells; TCR repertoire; acute myeloid leukemia (AML); chronic myelomonocytic leukemia (CMML); myelodysplastic neoplasms (MDS).
© 2025 The Author(s). European Journal of Immunology published by Wiley‐VCH GmbH.
MeSH terms
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Adult
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Aged
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Aged, 80 and over
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Azacitidine / pharmacology
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Azacitidine / therapeutic use
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Bone Marrow / immunology
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CD8-Positive T-Lymphocytes* / immunology
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Female
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Humans
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Leukemia, Myeloid, Acute* / drug therapy
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Leukemia, Myeloid, Acute* / immunology
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Leukemia, Myelomonocytic, Chronic* / drug therapy
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Leukemia, Myelomonocytic, Chronic* / immunology
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Male
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Middle Aged
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Myelodysplastic Syndromes* / drug therapy
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Myelodysplastic Syndromes* / immunology
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Receptors, Antigen, T-Cell* / genetics
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Receptors, Antigen, T-Cell* / immunology
Substances
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Azacitidine
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Receptors, Antigen, T-Cell