Abstract
Orthosteric inhibitors of the human heterodimeric DNA mismatch repair complex MutSβ were identified by high-throughput screening. Following extensive hit confirmation to remove false positives, two series were found to give consistent activity free of likely artefactual effects. Extensive hit profiling confirmed an ATP-competitive mode of action and resulted in our obtaining the first reported X-ray and cryo-EM structures of small molecule inhibitors of MutSβ occupying the ATP-binding site of MSH3.
Keywords:
ATPase; DNA mismatch repair; High-throughput screening; Huntington's disease; MSH3; MutSβ; Screening cascade.
Copyright © 2025. Published by Elsevier Ltd.
MeSH terms
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Adenosine Triphosphatases* / antagonists & inhibitors
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Adenosine Triphosphatases* / metabolism
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Adenosine Triphosphate / metabolism
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Binding Sites
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Cryoelectron Microscopy
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Crystallography, X-Ray
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DNA-Binding Proteins* / antagonists & inhibitors
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DNA-Binding Proteins* / chemistry
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DNA-Binding Proteins* / metabolism
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Enzyme Inhibitors* / chemical synthesis
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Enzyme Inhibitors* / chemistry
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Enzyme Inhibitors* / pharmacology
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High-Throughput Screening Assays
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Humans
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Models, Molecular
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Molecular Structure
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MutS Homolog 3 Protein / antagonists & inhibitors
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MutS Homolog 3 Protein / metabolism
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Small Molecule Libraries* / chemical synthesis
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Small Molecule Libraries* / chemistry
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Small Molecule Libraries* / pharmacology
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Steroids / chemistry
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Structure-Activity Relationship
Substances
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Adenosine Triphosphatases
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Adenosine Triphosphate
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DNA-Binding Proteins
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Enzyme Inhibitors
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MSH3 protein, human
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MutS Homolog 3 Protein
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Small Molecule Libraries
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Steroids