Novel environment exposure drives temporally defined and region-specific chromatin accessibility and gene expression changes in the hippocampus

Nat Commun. 2025 Aug 21;16(1):7787. doi: 10.1038/s41467-025-63029-6.

Abstract

Exposure to novel environments (NE) induces structural and functional changes in multiple brain areas, including the hippocampus, driven in part by changes in gene expression. However, the cell-type-specific transcriptional and chromatin responses to NE remain poorly understood. We employed single-nucleus multiomics and bulk RNA-seq of the hippocampal DG, CA3, and CA1 regions of male mice to profile gene expression and chromatin accessibility following NE exposure. We observed region-specific responses in excitatory neurons and diverse transcriptional changes in inhibitory and non-neuronal cells. NE-regulated genes were enriched for secreted factors, and their cell-type-specific receptor expression highlighted candidate signaling pathways involved in learning and memory. We identified thousands of cell-type-specific chromatin accessibility changes, with coordinated expression and accessibility patterns implicating FOS/AP-1 as a key regulator. These data provide a rich resource of chromatin accessibility and gene expression profiles across hippocampal cell types in response to NE, a physiological stimulus affecting learning and memory.

MeSH terms

  • Animals
  • CA1 Region, Hippocampal / metabolism
  • Chromatin* / genetics
  • Chromatin* / metabolism
  • Gene Expression Profiling
  • Gene Expression Regulation*
  • Hippocampus* / cytology
  • Hippocampus* / metabolism
  • Male
  • Memory / physiology
  • Mice
  • Mice, Inbred C57BL
  • Neurons / metabolism
  • Proto-Oncogene Proteins c-fos / genetics
  • Proto-Oncogene Proteins c-fos / metabolism
  • RNA-Seq
  • Transcription Factor AP-1 / genetics
  • Transcription Factor AP-1 / metabolism
  • Transcriptome

Substances

  • Chromatin
  • Proto-Oncogene Proteins c-fos
  • Transcription Factor AP-1