Abstract
Neuroinflammation contributes to Alzheimer's disease (AD), but the molecules and pathways that initiate inflammation are unclear. In this issue of Immunity, Song et al. demonstrate that ZBP1-RIPK1 signaling in microglia can drive AD, wherein ZBP1 is activated by left-handed Z-DNA leaking from mitochondria, presenting new molecular targets for AD therapeutics.
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MeSH terms
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Alzheimer Disease* / immunology
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Alzheimer Disease* / metabolism
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Animals
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DNA-Binding Proteins* / metabolism
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Humans
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Inflammation / immunology
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Mice
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Microglia* / immunology
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Microglia* / metabolism
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Mitochondria / metabolism
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RNA-Binding Proteins* / metabolism
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Receptor-Interacting Protein Serine-Threonine Kinases* / metabolism
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Signal Transduction
Substances
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DNA-Binding Proteins
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Receptor-Interacting Protein Serine-Threonine Kinases
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RNA-Binding Proteins