Germline epigenome editing identifies H3K9me3 as a mediator of intergenerational DNA methylation recovery in mice

Nat Commun. 2025 Dec 25;16(1):11200. doi: 10.1038/s41467-025-67488-9.

Abstract

Inter/transgenerational epigenetic inheritance is a crucial and controversial theory that could reshape the concept of genetics. To investigate this theory directly, we invent a system for targeted reprogramming of epigenetic memory in mouse sperm. Using this system, we erase DNA methylation at the differentially methylated region of the H19 gene (H19-DMR) in sperm, which causes Silver-Russell syndrome-like phenotypes in F1 offspring. Although DNA methylation is fully lost in the sperm, it is partially restored during pre-implantation development, suggesting the existence of epigenetic memory that instructs de novo DNA methylation. Importantly, targeted removal of histone modifications in zygotes reveals that tri-methylation at lysine 9 of histone H3 (H3K9me3), which is deposited shortly after fertilization, is required for the subsequent de novo DNA methylation at the H19-DMR. Thus, our study provides a robust germline editing tool, which reveals partial intergenerational inheritance and no transgenerational inheritance at the model locus. Furthermore, we identify H3K9me3 as a mediator for DNA methylation recovery also acting at imprinted loci.

MeSH terms

  • Animals
  • DNA Methylation* / genetics
  • Epigenesis, Genetic*
  • Epigenome Editing
  • Epigenome* / genetics
  • Female
  • Gene Editing* / methods
  • Genomic Imprinting
  • Germ Cells / metabolism
  • Histones* / genetics
  • Histones* / metabolism
  • Male
  • Mice
  • Mice, Inbred C57BL
  • RNA, Long Noncoding / genetics
  • RNA, Long Noncoding / metabolism
  • Spermatozoa / metabolism
  • Zygote / metabolism

Substances

  • Histones
  • H19 long non-coding RNA
  • RNA, Long Noncoding