Tools to Quantify and Characterize the Persistent Reservoir in People with HIV-1: Focus on Non-B Subtypes

Viruses. 2026 Jan 14;18(1):110. doi: 10.3390/v18010110.

Abstract

Human immunodeficiency virus type 1 (HIV-1) continues to be a major global health burden. Combination antiretroviral therapy (cART) effectively abrogates HIV-1 replication and has transformed HIV-1 infection from a fatal to chronic disease. While ART can suppress viremia to undetectable levels in people living with HIV-1 (PWH), a small reservoir of cells infected with replication-competent HIV-1 persists and can lead to viral rebound upon ART interruption. This persistent HIV-1 reservoir can be quantified and characterized by measuring replication of infectious HIV-1 using a quantitative viral outgrowth assay (qVOA), or by measuring HIV-1 DNA, RNA, or protein levels as a proxy for the reservoir. Tools to quantify the reservoir in these distinct molecular compartments have been developed for HIV-1 subtype B, which is predominant in the Global North. However, non-B subtypes constitute the majority of HIV-1 infections worldwide. Here, we discuss the wide range of reservoir quantitation and characterization tools, explore their limitations, and, where applicable, their adaptations to non-B subtypes. We conclude that standardized tools should be used to characterize reservoir dynamics of HIV-1 B and non-B subtypes. These tests should be well-validated and accessible to all laboratories world-wide to be able to draw conclusions about subtype-specific reservoir dynamics.

Keywords: HIV-1; HIV-1 reservoir; Non-B subtypes; reservoir quantitation.

Publication types

  • Review

MeSH terms

  • DNA, Viral
  • Disease Reservoirs / virology
  • HIV Infections* / drug therapy
  • HIV Infections* / virology
  • HIV-1* / classification
  • HIV-1* / genetics
  • HIV-1* / isolation & purification
  • HIV-1* / physiology
  • Humans
  • Persistent Infection / virology
  • RNA, Viral
  • Viral Load
  • Virus Latency
  • Virus Replication

Substances

  • RNA, Viral
  • DNA, Viral