Incidence and outcomes of atrial arrhythmia with cyclin dependent kinase 4/6 inhibitors in hormone receptor-positive / human epidermal growth factor receptor 2-negative breast cancer

Int J Cardiol. 2026 Jul 1:454:134466. doi: 10.1016/j.ijcard.2026.134466. Epub 2026 Mar 27.

Abstract

Background: Cyclin-dependent kinase 4/6 (CDK4/6) inhibitors are increasingly used in hormone receptor-positive, HER2-negative (HR+/HER2-) breast cancer, yet emerging data suggest potential cardiotoxicity, including atrial arrhythmias (AA). Understanding incidence and outcomes of AA is essential as indications for CDK4/6 inhibitors expand.

Objectives: To evaluate the incidence of new-onset AA and associated outcomes in patients with HR+/HER2- breast cancer treated with CDK4/6 inhibitors.

Methods: We conducted a retrospective cohort study of patients who received CDK4/6 inhibitors for HR+/HER2- breast cancer at Mayo Clinic from 2015 to 2024. The primary outcome was incidence of AA (atrial fibrillation, atrial flutter, or atrial tachycardia). Secondary outcomes included cerebrovascular events and all-cause mortality. Fine-Gray subdistribution hazard models and Cox regression models were used to assess risk factors for AA and all-cause mortality, respectively.

Results: Among 2773 patients, 59% received palbociclib, 28% abemaciclib, and 14% ribociclib. New-onset AA occurred in 42, cumulative incidence at 5 years of 2.1% (95% CI 1.5-2.8%). No significant differences in 5-year AA incidence were observed between agents: 1.5% (95% CI 0.7-2.8%), 2.3 (95% CI 1.6-3.2%), 1.0 (95% CI 0.3-2.6%) (p = 0.49). Multivariable analysis identified age at treatment as the only independent predictor of AA (HR 1.07, 95% CI 1.04-1.09, p < 0.001). New-onset AA was associated with increased mortality (HR 1.56, 95% CI 1.10-2.20, p = 0.012).

Conclusions: Patients who receive CDK4/6 inhibitors for breast cancer experience a low but measurable risk of new-onset atrial arrhythmias. There was no significant difference in new-onset AA risk between agents. Prospective studies are needed to define mechanisms and guide monitoring strategies.

Keywords: Arrhythmia; Atrial arrhythmia; Breast cancer; Cardio-oncology; Cardiotoxicity; Risk stratification.

MeSH terms

  • Aged
  • Aminopyridines / adverse effects
  • Atrial Fibrillation* / chemically induced
  • Atrial Fibrillation* / epidemiology
  • Benzimidazoles / adverse effects
  • Breast Neoplasms* / drug therapy
  • Breast Neoplasms* / metabolism
  • Cohort Studies
  • Cyclin-Dependent Kinase 4* / antagonists & inhibitors
  • Cyclin-Dependent Kinase 6* / antagonists & inhibitors
  • Erb-b2 Receptor Tyrosine Kinases* / metabolism
  • Female
  • Humans
  • Incidence
  • Middle Aged
  • Piperazines / adverse effects
  • Protein Kinase Inhibitors* / adverse effects
  • Protein Kinase Inhibitors* / therapeutic use
  • Purines / adverse effects
  • Pyridines / adverse effects
  • Receptors, Estrogen / metabolism
  • Receptors, Progesterone / metabolism
  • Retrospective Studies
  • Treatment Outcome

Substances

  • Cyclin-Dependent Kinase 6
  • Erb-b2 Receptor Tyrosine Kinases
  • Cyclin-Dependent Kinase 4
  • Pyridines
  • Piperazines
  • Protein Kinase Inhibitors
  • palbociclib
  • Purines
  • ERBB2 protein, human
  • Benzimidazoles
  • Aminopyridines
  • CDK4 protein, human
  • CDK6 protein, human
  • ribociclib
  • abemaciclib
  • Receptors, Estrogen
  • Receptors, Progesterone