Ribonuclease DIS3 delays aging and senescence by generating tRNA halves

Nat Commun. 2026 May 21;17(1):6698. doi: 10.1038/s41467-026-73295-7.

Abstract

Transfer RNA (tRNA) halves (tRHs) are generated via the cleavage of tRNAs, but their roles in aging and longevity remain poorly understood. Here, we demonstrate a direct role of tRHs in aging in metazoans. Through a genetic screen using Caenorhabditis elegans, we identify DIS-3/DIS3 as a ribonuclease that catalyzes tRH generation, including 5'-tRH-Gln and 5'-tRH-Asp, from tRNAs. Among them, 5'-tRH-Gln is essential for longevity conferred by various interventions, including dietary restriction. Generation of 5'-tRH-Gln reduces translation via ribosomal protein binding and upregulates the SKN-1/NRF transcription factor responsible for lifespan extension. We further show that mammalian DIS3 contributes to tRH generation and delays cellular senescence through translation downregulation by another tRH, 5'-tRH-Cys. Overall, our data demonstrate that DIS-3/DIS3 is an evolutionarily conserved tRH-generating ribonuclease that counteracts organismal and cellular aging.

MeSH terms

  • Aging* / genetics
  • Animals
  • Caenorhabditis elegans Proteins* / genetics
  • Caenorhabditis elegans Proteins* / metabolism
  • Caenorhabditis elegans* / genetics
  • Caenorhabditis elegans* / metabolism
  • Caenorhabditis elegans* / physiology
  • Cellular Senescence / genetics
  • Humans
  • Longevity / genetics
  • RNA, Transfer* / genetics
  • RNA, Transfer* / metabolism
  • Ribonucleases* / genetics
  • Ribonucleases* / metabolism

Substances

  • RNA, Transfer
  • Caenorhabditis elegans Proteins
  • Ribonucleases