Investigating the amyloid-tau-neurodegeneration framework in Alzheimer's disease using semi-supervised multimodal imaging data fusion

Alzheimers Dement (Amst). 2026 May 21;18(2):e70360. doi: 10.1002/dad2.70360. eCollection 2026 Apr-Jun.

Abstract

Introduction: Alzheimer's disease (AD) heterogeneity complicates diagnosis and prognosis. Uncovering amyloid-tau-neurodegeneration (A-T-N) patterns may improve diagnostic prediction.

Methods: We applied SuperBigFLICA (SBF), a semi-supervised multimodal fusion method, to gray matter density, cortical thickness (CT), pial surface area, amyloid and tau positron emission tomography maps from 274 Alzheimer's Disease Neuroimaging Initiative 3 participants to derive 50 latent components predictive of cognitive decline. Subject loadings were then used to predict diagnosis (cognitively normal, mild cognitive impairment, dementia) and apolipoprotein E (APOE) ε4 status via least absolute shrinkage and selection operator logistic regression, compared to demographic, single-modality, and naïve fusion comparator models.

Results: SBF modestly predicted out-of-sample concurrent clinical severity (Clinical Dementia Rating Sum of Boxes; r = 0.21), yet models using SBF-derived loadings were among the strongest comparator models (area under the receiver operating characteristic curve; = 0.80 for diagnosis; 0.83 for APOE ε4). Amyloid alterations in sensory areas best separated dementia, while a tri-modal tau-neurodegeneration pattern related to disease progression. Loadings were validated through cerebrospinal fluid correlations.

Discussion: SBF improves prediction and reveals interpretable patterns that better classify clinical diagnoses and APOE ε4 than traditional approaches.

Keywords: Alzheimer's disease; amyloid–tau–neurodegeneration framework; latent components; multimodal data fusion; multimodal neuroimaging; semi‐supervised learning.