Abstract
The polyoma ts-a function was investigated by using an in vitro DNA-synthesizing system. A comparison of systems derived from ts25 (a ts-a group mutant)-and ts1260 (a late group mutant)-infected cells showed that the activation energies for DNA chain elongation and the mechanisms of discontinous growth were identical for both mutants.
MeSH terms
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Animals
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Cell Line
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Centrifugation, Density Gradient
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Cytosine Nucleotides / metabolism
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DNA / biosynthesis
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DNA Replication
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DNA, Viral / biosynthesis*
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Mice
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Mutation*
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Phosphorus Radioisotopes
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Polyomavirus / growth & development
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Polyomavirus / metabolism*
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Temperature
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Thymidine / metabolism
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Time Factors
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Tritium
Substances
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Cytosine Nucleotides
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DNA, Viral
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Phosphorus Radioisotopes
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Tritium
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DNA
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Thymidine