Galactosamine hepatitis: key role of the nucleotide deficiency period in the pathogenesis of cell injury and cell death

Rev Physiol Biochem Pharmacol. 1974;(71):77-106. doi: 10.1007/BFb0027661.
No abstract available

Publication types

  • Review

MeSH terms

  • Animals
  • Blood Coagulation Factors / metabolism
  • Carbohydrates / biosynthesis
  • Carcinoma, Hepatocellular / metabolism
  • Cell Survival
  • Chemical and Drug Induced Liver Injury / enzymology
  • Chemical and Drug Induced Liver Injury / metabolism*
  • Chemical and Drug Induced Liver Injury / pathology
  • Deoxy Sugars / pharmacology
  • Disease Models, Animal
  • Galactosamine* / metabolism
  • Galactosamine* / pharmacology
  • Galactose / pharmacology
  • Glucosamine / pharmacology
  • Hyperbilirubinemia / chemically induced
  • Liver Neoplasms
  • Macromolecular Substances
  • Protein Biosynthesis
  • Pyrimidine Nucleotides / pharmacology
  • RNA / biosynthesis
  • Time Factors
  • Uracil Nucleotides / deficiency
  • Uracil Nucleotides / metabolism
  • Uridine / pharmacology
  • Uridine Diphosphate Sugars / metabolism

Substances

  • Blood Coagulation Factors
  • Carbohydrates
  • Deoxy Sugars
  • Macromolecular Substances
  • Pyrimidine Nucleotides
  • Uracil Nucleotides
  • Uridine Diphosphate Sugars
  • RNA
  • Galactosamine
  • Glucosamine
  • Uridine
  • Galactose