Clinical, metabolic and molecular consequences of genetic disorders of the pentose phosphate pathway
- PMID: 4909475
- PMCID: PMC1811287
Clinical, metabolic and molecular consequences of genetic disorders of the pentose phosphate pathway
Similar articles
-
Red cell enzyme deficiencies as a cause of hemolytic disorders.Annu Rev Med. 1972;23:93-100. doi: 10.1146/annurev.me.23.020172.000521. Annu Rev Med. 1972. PMID: 4268540 Review. No abstract available.
-
[Laboratory diagnosis of enzymopenic hemolytic anemia].Pediatria (Napoli). 1972;80(2):186-97. Pediatria (Napoli). 1972. PMID: 4262752 Review. Italian. No abstract available.
-
Hemolytic anemia in hereditary pyrimidine 5'-nucleotidase deficiency: nucleotide inhibition of G6PD and the pentose phosphate shunt.Blood. 1982 Nov;60(5):1212-8. Blood. 1982. PMID: 6289944
-
[Hereditary enzymopenic anemia].Minerva Med. 1972 Dec 1;63(86):4721-3. Minerva Med. 1972. PMID: 4264634 Italian. No abstract available.
-
Metabolism of the hexose monophosphate shunt in glucose-6-phosphate dehydrogenase deficiency and closely interrelated reactions.Haematologia (Budap). 1982 Dec;15(4):401-7. Haematologia (Budap). 1982. PMID: 7186479
Cited by
-
Newborn Screening for G6PD Deficiency in Xiamen, China: Prevalence, Variant Spectrum, and Genotype-Phenotype Correlations.Front Genet. 2021 Oct 1;12:718503. doi: 10.3389/fgene.2021.718503. eCollection 2021. Front Genet. 2021. PMID: 34659341 Free PMC article.
-
Stimulability, adenosine triphosphatases and their control by cellular redox processes.Naturwissenschaften. 1971 Sep;58(9):439-43. doi: 10.1007/BF00624617. Naturwissenschaften. 1971. PMID: 4255843 Review. No abstract available.
References
Publication types
MeSH terms
Substances
LinkOut - more resources
Full Text Sources