Monoclonal antibodies to the thyrotropin receptor: implications for receptor structure and the action of autoantibodies in Graves disease

Proc Natl Acad Sci U S A. 1981 May;78(5):3180-4. doi: 10.1073/pnas.78.5.3180.

Abstract

Hybridoma cells have been obtained by fusing P3-NS1/1-Ag4-1 mouse myeloma cells with spleen cells from mice immunized with solubilized preparations of the thyrotropin receptor. Five clones were produced that secrete a monoclonal antibody whose binding to thyroid membranes is specifically inhibited by unlabeled thyrotropin. The antibody interacts with functioning thyroid cells in culture but not with nonfunctioning cells; this interaction is prevented by thyrotropin. The antibodies are capable of competitively blocking thyrotropin binding to bovine thyroid membrane preparations; they prevent 125I-labeled thyrotropin binding to a solubilized preparation of the glycoprotein component of the bovine thyrotropin receptor but are unable to inhibit 125I-labeled thyrotropin binding to liposomes containing gangliosides at comparable concentrations. They prevent 125I-labeled thyrotropin binding to rat, bovine, or human (Graves disease) thyroid membrane preparations. They do not stimulate adenylate cyclase activity in thyroid membrane preparations but can inhibit thyrotropin-stimulated iodide uptake by functioning thyroid cells in culture.

MeSH terms

  • Animals
  • Antibodies*
  • Antibodies, Monoclonal
  • Antigens, Surface / analysis
  • Autoantibodies*
  • Binding, Competitive
  • Cell Line
  • Cholera Toxin / pharmacology
  • Clone Cells
  • Graves Disease / metabolism*
  • Humans
  • Hybrid Cells / immunology
  • Kinetics
  • Mice
  • Plasmacytoma
  • Receptors, Cell Surface / drug effects
  • Receptors, Cell Surface / metabolism*
  • Receptors, Thyrotropin
  • Spleen / immunology
  • Thyrotropin / metabolism*

Substances

  • Antibodies
  • Antibodies, Monoclonal
  • Antigens, Surface
  • Autoantibodies
  • Receptors, Cell Surface
  • Receptors, Thyrotropin
  • Thyrotropin
  • Cholera Toxin