Ketoconazole blocks testosterone synthesis

Arch Intern Med. 1982 Nov;142(12):2137-40.

Abstract

Ketoconazole, a new oral drug used to treat systemic and superficial mycoses, inhibits sterol synthesis in fungi. The development of gynecomastia in two patients prompted us to investigate the effect of the drug on testosterone production. After a 200-, 400-, or 600-mg dose, volunteer male testosterone serum concentrations fell markedly, but returned toward baseline eight to 24 hours later as ketoconazole serum concentrations waned. A marked but transient drop in testosterone levels occurred in patients receiving long-term therapy, and continuous testosterone depression was noted in one. A block of synthesis was demonstrated in vitro. Ketoconazole at concentrations achievable in serum with currently used doses blocked basal and gonadotropin-stimulated testosterone production by rat Leydig cells. The diminution of testosterone synthesis could be significant as further therapeutic trials may use larger doses or more than once-daily administration. The paucity of reports of endocrinologic toxicity may relate to the "escape" from the block demonstrated in vivo.

Publication types

  • Case Reports
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adult
  • Animals
  • Antifungal Agents / adverse effects
  • Antifungal Agents / pharmacology*
  • Coccidioidomycosis / drug therapy
  • Gynecomastia / chemically induced
  • Humans
  • Imidazoles / adverse effects
  • Imidazoles / pharmacology*
  • Ketoconazole
  • Leydig Cells / drug effects
  • Male
  • Piperazines / adverse effects
  • Piperazines / pharmacology*
  • Rats
  • Rats, Inbred Strains
  • Testosterone / antagonists & inhibitors*
  • Testosterone / biosynthesis

Substances

  • Antifungal Agents
  • Imidazoles
  • Piperazines
  • Testosterone
  • Ketoconazole