Abstract
Palmitoyl-CoA, at the concentrations which did cause uncoupling of oxidative phosphorylation, decreased the energy pool of liver mitochondria in presence of oligomycine. Characteristic ability of palmitoyl-CoA to increase the proton transport in mitochondrial membranes was responsible for the deenergizing action.
MeSH terms
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Acyl Coenzyme A / pharmacology*
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Animals
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Female
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Kinetics
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Mitochondria, Liver / drug effects
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Mitochondria, Liver / metabolism*
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Mitochondrial ADP, ATP Translocases / metabolism*
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Nucleotidyltransferases / metabolism*
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Oligomycins / pharmacology
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Oxidative Phosphorylation / drug effects
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Palmitoyl Coenzyme A / pharmacology*
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Rats
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Submitochondrial Particles / drug effects
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Submitochondrial Particles / metabolism
Substances
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Acyl Coenzyme A
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Oligomycins
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Palmitoyl Coenzyme A
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Mitochondrial ADP, ATP Translocases
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Nucleotidyltransferases