Pathogenesis of herpes simplex labialis: excretion of virus in the oral cavity

J Clin Microbiol. 1984 May;19(5):675-9. doi: 10.1128/jcm.19.5.675-679.1984.

Abstract

Excretion of herpes simplex virus (HSV) in the oral cavity was studied in eight human subjects with a history of herpes labialis. Serial intraoral specimens were obtained by gargling broth and examined for virus by centrifugal inoculation of primary human amnion cells. Forty-seven of 637 specimens (7.4%) contained HSV. The majority of isolates (62%) were found in clusters, and the rate of excretion was significantly increased during the common cold (21%) and after oral trauma (17%) (P = 0.001 and 0.04, respectively). Oral HSV excretion often occurred in parallel with episodes of herpes labialis but could not be attributed to viral contamination from a labial lesion. Each patient excreted only one strain of HSV type 1 as determined by restriction endonuclease analysis with KpnI and BamHI. Unexpectedly, prodromal symptoms of herpes labialis were commonly not followed by development of a lesion (false prodrome). False prodromes were associated with a high rate of oral HSV excretion (60%). Intraoral ulcers on the gingivae and hard palate were frequently associated with oral HSV excretion (31%) and are the most likely source of HSV in the oral cavity.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adult
  • DNA Restriction Enzymes
  • DNA, Viral / analysis
  • Female
  • Herpes Labialis / microbiology*
  • Herpes Labialis / pathology
  • Humans
  • Life Change Events
  • Male
  • Middle Aged
  • Mouth / microbiology*
  • Simplexvirus / genetics
  • Simplexvirus / isolation & purification
  • Simplexvirus / pathogenicity*
  • Stomatitis, Herpetic / microbiology*
  • Stomatitis, Herpetic / pathology

Substances

  • DNA, Viral
  • DNA Restriction Enzymes