Energy depletion retards the loss of membrane transport during reticulocyte maturation

Proc Natl Acad Sci U S A. 1983 Aug;80(16):4978-82. doi: 10.1073/pnas.80.16.4978.

Abstract

The effect of metabolic depletion on the maturation-associated loss of membrane functions has been studied by using sheep reticulocytes incubated in vitro at 37 degrees C for periods up to 41 hr. ATP was either maintained with glucose, adenosine plus inosine, or depleted with 2-deoxyglucose plus arsenate. Two membrane transport systems were studied: Na+-dependent glycine transport activity and the sodium pump, estimated from measurements of the number of [3H]ouabain binding sites per cell. Both transport systems were decreased during maturation. However, the decrease was much less in ATP-depleted cells compared to ATP-replete cells. It is concluded that the loss of certain functions during reticulocyte maturation is retarded by metabolic depletion.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenosine Triphosphate / blood*
  • Animals
  • Biological Transport / drug effects
  • Cells, Cultured
  • Erythrocyte Aging*
  • Erythrocyte Membrane / metabolism*
  • Erythrocytes / metabolism*
  • Glycine / blood*
  • Ouabain / blood
  • Receptors, Drug / metabolism
  • Reticulocytes / metabolism*
  • Sheep
  • Sodium / pharmacology
  • Sodium-Potassium-Exchanging ATPase*

Substances

  • Receptors, Drug
  • cardiac glycoside receptors
  • Ouabain
  • Adenosine Triphosphate
  • Sodium
  • Sodium-Potassium-Exchanging ATPase
  • Glycine