Acceleration of cytokinesis in PtK1 cells treated with microtubule inhibitors

Exp Cell Res. 1983 Apr 1;144(2):345-51. doi: 10.1016/0014-4827(83)90413-5.

Abstract

Mitotic PtK1 cells were treated at furrow initiation with microtubule poisons to determine the role of microtubules in the regulation of terminal cytokinetic events. The administration of anti-microtubule agents in late anaphase accelerated the rate of cytokinesis by approx. 60% as measured by changes in furrow diameter. Application of colcemid, nocodazole, or vinblastine sulfate to cells at furrow initiation all increased the rate of furrowing. Nocodazole, applied at various concentrations, demonstrated a dose-dependent relationship with furrowing rate. These results suggest a coupling between the disorganization and depolymerization of microtubules and the acceleration of furrowing. Electron microscopic analysis of cells treated with microtubule inhibitors show approx. 70% fewer microtubule profiles in the constricted region between the daughter cells. Microtubules may play a restraining role in the rate of furrowing.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Anaphase
  • Animals
  • Benzimidazoles / pharmacology*
  • Cell Division* / drug effects
  • Cell Line
  • Cytoskeleton / ultrastructure
  • Demecolcine / pharmacology*
  • Dose-Response Relationship, Drug
  • Kinetics
  • Macropodidae
  • Microtubules / physiology*
  • Microtubules / ultrastructure
  • Nocodazole
  • Vinblastine / pharmacology*

Substances

  • Benzimidazoles
  • Vinblastine
  • Nocodazole
  • Demecolcine