Differential inhibition of multiple forms of DNA polymerase alpha from IMR-32 human neuroblastoma cells

Proc Natl Acad Sci U S A. 1981 May;78(5):2683-7. doi: 10.1073/pnas.78.5.2683.

Abstract

Three forms of DNA polymerase (pol) alpha from human neuroblastoma IMR-32 were separated by DEAE column chromatography. All sedimented at approximately 7 S in 5-20% continuous sucrose density gradients. All were heat labile, with pol alpha 2 the most (90% inactivated) and pol alpha 3 the least (50% inactivated) sensitive to heating for 5 min at 50 degrees C. pol alpha 1 and alpha 2 efficiently utilized activated calf thymus DNA as template. The most active form, pol alpha 2, used both poly(dA).(dT)12-18 and poly(dT).(dA)12-18 as template at equal rates. Differential inhibition of DNA polymerase alpha activities was examined in the presence of ricin, hemin, and a nonhistone chromatin protein. All three polymerases were inhibited by both ricin (nonreduced) and hemin, with pol alpha 2 the most (80-90%) and pol alpha 3 the least (60%) sensitive in each case. In contrast, only pol alpha 2 and alpha 3 activities were inhibited (80-85%) by rat liver nonhistone chromatin protein.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Cations, Divalent
  • Cell Line
  • Chromosomal Proteins, Non-Histone / isolation & purification
  • DNA Polymerase II / antagonists & inhibitors*
  • DNA Polymerase II / isolation & purification
  • Humans
  • Isoenzymes / antagonists & inhibitors*
  • Isoenzymes / isolation & purification
  • Kinetics
  • Lectins / pharmacology
  • Liver / analysis
  • Neuroblastoma / enzymology*
  • Nucleic Acid Synthesis Inhibitors*
  • Polydeoxyribonucleotides
  • Rats
  • Templates, Genetic
  • Toxins, Biological / pharmacology

Substances

  • Cations, Divalent
  • Chromosomal Proteins, Non-Histone
  • Isoenzymes
  • Lectins
  • Nucleic Acid Synthesis Inhibitors
  • Polydeoxyribonucleotides
  • Toxins, Biological
  • DNA Polymerase II