Evidence in man that cigarette smoking induces vasopressin release via an airway-specific mechanism

J Clin Endocrinol Metab. 1980 Jul;51(1):170-2. doi: 10.1210/jcem-51-1-170.

Abstract

The plasma vasopressin response to intravenous nicotine (2 mg) and smoking cigarettes of high (1.2 mg) and low (0.6 mg) nicotine content was studied in healthy young subjects with a history of cigarette smoking. Nicotine administered intravenously had no effect on plasma vasopressin. There was a statistically significant increase in plasma vasopressin after both the low and high content cigarettes, with the high content cigarettes resulting in a significantly greater increase. All three interventions resulted in transient increases in blood pressure and pulse of similar magnitude. These studies indicate that cigarette smoking-induced vasopressin release is not mediated via an action of circulating nicotine, and suggest the presence of an airway-specific mechanism for vasopressin release.

Publication types

  • Comparative Study
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adult
  • Humans
  • Lung / drug effects
  • Nicotine / administration & dosage
  • Nicotine / pharmacology*
  • Smoking*
  • Vasopressins / blood*

Substances

  • Vasopressins
  • Nicotine