Mechanism of postprandial hyperglycaemia in diabetes mellitus

Eur J Gastroenterol Hepatol. 1995 Aug;7(8):724-9.

Abstract

Abnormalities of hepatic and extrahepatic tissues combine to produce the postprandial hyperglycaemia characteristic of non-insulin-dependent diabetes mellitus. The altered metabolic responses are primarily due to defects in insulin secretion and action. Other regulators of carbohydrate metabolism also contribute to the altered milieu.

Publication types

  • Review

MeSH terms

  • Diabetes Mellitus, Type 2 / complications
  • Diabetes Mellitus, Type 2 / metabolism
  • Diabetes Mellitus, Type 2 / physiopathology*
  • Dietary Carbohydrates / metabolism
  • Eating
  • Humans
  • Hyperglycemia / etiology
  • Hyperglycemia / physiopathology*

Substances

  • Dietary Carbohydrates