Abundant but inactive-state gp140proto-trk is expressed in neuroblastomas of patients with good prognosis

Jpn J Cancer Res. 1994 Jan;85(1):32-9. doi: 10.1111/j.1349-7006.1994.tb02883.x.

Abstract

Steady-state levels of gp140proto-trk in cell lines and tumor tissues of neuroblastoma were examined by immunoblotting with anti-gp140proto-trk. The level of gp140proto-trk varied but showed good correlations with the stage of the tumor and the age of the patients at the time of diagnosis. Moreover, patients with higher expression of gp140proto-trk clearly had a far better survival rate than those with lower expression, suggesting that suppression of gp140proto-trk strongly correlates with the malignant conversion of the tumor. However, we found that neither autophosphorylation of gp140proto-trk nor tyrosine phosphorylation of cellular proteins was elevated in tumors of the higher expression group. These results suggest that gp140proto-trk does not actively participate in the process of transformation or the suppression of malignant conversion. Rather, the higher level of gp140proto-trk may reflect the greater level of differentiation of tumor cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Age Factors
  • Child
  • Child, Preschool
  • Glioma / chemistry
  • Humans
  • Infant
  • Neoplasm Staging
  • Neuroblastoma / chemistry*
  • Neuroblastoma / mortality
  • Neuroblastoma / pathology
  • Phosphorylation
  • Phosphotyrosine
  • Prognosis
  • Proto-Oncogene Proteins / analysis*
  • Proto-Oncogene Proteins / metabolism
  • Receptor Protein-Tyrosine Kinases / analysis*
  • Receptor Protein-Tyrosine Kinases / metabolism
  • Receptor, trkA
  • Receptors, Nerve Growth Factor / analysis*
  • Receptors, Nerve Growth Factor / metabolism
  • Tumor Cells, Cultured
  • Tyrosine / analogs & derivatives
  • Tyrosine / analysis

Substances

  • Proto-Oncogene Proteins
  • Receptors, Nerve Growth Factor
  • Phosphotyrosine
  • Tyrosine
  • Receptor Protein-Tyrosine Kinases
  • Receptor, trkA