Abstract
Overproduction of TmrB protein, a 22.5-kDa protein with an N-terminal ATP-binding region and a C-terminal amphiphilic alpha-helix, confers resistance to tunicamycin on Bacillus subtilis. TmrB protein was found to bind Sepharose 6B to which tunicamycin was covalently linked. Experiments with mutant proteins found that the C-terminal region of TmrB protein might be involved in the binding to tunicamycin.
Publication types
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Comparative Study
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Research Support, Non-U.S. Gov't
MeSH terms
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Bacillus subtilis / metabolism*
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Bacterial Proteins / biosynthesis
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Bacterial Proteins / chemistry
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Bacterial Proteins / genetics
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Bacterial Proteins / metabolism*
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Binding Sites
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Binding, Competitive
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Blotting, Western
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Chemical Fractionation
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Drug Resistance, Microbial
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Electrophoresis, Polyacrylamide Gel
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Gene Expression Regulation, Bacterial / genetics
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Membrane Proteins / biosynthesis
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Membrane Proteins / chemistry
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Membrane Proteins / genetics
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Membrane Proteins / metabolism*
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Molecular Weight
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Mutation
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Precipitin Tests
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Tunicamycin / metabolism*
Substances
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Bacterial Proteins
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Membrane Proteins
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Tunicamycin
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tmrB protein, Bacillus subtilis