Apolipoprotein B mRNA editing in vitro is a zinc-dependent process

Biochem Biophys Res Commun. 1993 Dec 30;197(3):1410-4. doi: 10.1006/bbrc.1993.2634.

Abstract

Apolipoprotein B mRNA editing involves the conversion of a cytidine at nucleotide 6666 to a uridine, thereby creating a translational stop codon from a glutamine codon. Recent evidence suggest that a cytidine deaminase is responsible for catalyzing the C to U conversion. All known nucleotide deaminases require zinc as part of their catalytic domain. Utilizing the selective chelator for zinc, 1,10-penanthroline, we demonstrate that in vitro editing activity in rat liver extracts is zinc dependent. These data, taken together with recent reports, strongly support a role for a cytidine deaminase and nucleotide conversion as the catalytic mechanism for apoB mRNA editing.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Apolipoproteins B / biosynthesis*
  • Cell Nucleus / metabolism*
  • Cytidine Deaminase / metabolism
  • Iron Chelating Agents / pharmacology
  • Liver / drug effects
  • Liver / metabolism*
  • Phenanthrolines / pharmacology
  • RNA, Messenger / isolation & purification
  • RNA, Messenger / metabolism*
  • Rats
  • Salts / pharmacology
  • Zinc / metabolism
  • Zinc / pharmacology*

Substances

  • Apolipoproteins B
  • Iron Chelating Agents
  • Phenanthrolines
  • RNA, Messenger
  • Salts
  • Cytidine Deaminase
  • Zinc
  • 1,10-phenanthroline