Epstein-Barr virus infection is associated with increased apoptosis in untreated and phorbol ester-treated human Burkitt's lymphoma (AW-Ramos) cells

Biochem Biophys Res Commun. 1993 May 14;192(3):1415-23. doi: 10.1006/bbrc.1993.1574.

Abstract

Several papers have recorded that Epstein-Barr virus (EBV) infection can protect B-cells from apoptosis. In the present paper, we record an increase in apoptosis in an EBV-infected human Burkitt's lymphoma cell line (AW-Ramos), compared with its virus free counterpart (Ramos). The viral-infected cells died more rapidly by apoptosis during normal growth in culture: at 72h, Ramos cells had 8% apoptosis and AW-Ramos had 27% apoptosis. The EBV-infected cells were particularly sensitive to treatment with phorbol-12, 13-dibutyrate (PDBu, 0.05 microgram/ml): at 72h, Ramos cells had 12% apoptosis and AW-Ramos had 42% apoptosis. DNA gel electrophoresis supported the morphological findings. Our results serve as a caveat against generalizations that may be made about prevention of apoptosis by EBV infection.

MeSH terms

  • Apoptosis* / drug effects
  • Burkitt Lymphoma / pathology
  • Burkitt Lymphoma / ultrastructure
  • Cell Division
  • Cell Survival
  • Cell Transformation, Viral*
  • DNA, Neoplasm / analysis
  • Herpesvirus 4, Human / genetics*
  • Humans
  • Kinetics
  • Microscopy, Electron
  • Necrosis
  • Phorbol 12,13-Dibutyrate / pharmacology*
  • Time Factors
  • Tumor Cells, Cultured

Substances

  • DNA, Neoplasm
  • Phorbol 12,13-Dibutyrate