Kappa opioid receptor-mediated regulation of prolactin and alpha-melanocyte-stimulating hormone secretion in male and female rats

Life Sci. 1993;53(10):795-801. doi: 10.1016/0024-3205(93)90501-s.

Abstract

The purpose of this study was to examine the effects of the kappa opioid receptor agonist U-50,488 and antagonist nor-binaltorphimine (NOR-BNI) on the secretion of prolactin and alpha-melanocyte-stimulating hormone (alpha MSH) in male and female rats. Activation of kappa opioid receptors with U-50,488 increased plasma prolactin concentrations in both male and female rats, whereas blockade of kappa opioid receptors with NOR-BNI decreased plasma prolactin concentrations in male, but not in female rats. U-50,488 also increased and NOR-BNI decreased plasma concentrations of alpha MSH; similar effects were observed in both male and female rats. These results reveal a sexual difference in kappa opioid receptor-mediated regulation of prolactin, but not alpha MSH secretion.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • 3,4-Dichloro-N-methyl-N-(2-(1-pyrrolidinyl)-cyclohexyl)-benzeneacetamide, (trans)-Isomer
  • Animals
  • Female
  • Male
  • Naltrexone / analogs & derivatives
  • Naltrexone / pharmacology
  • Prolactin / blood
  • Prolactin / metabolism*
  • Pyrrolidines / pharmacology
  • Rats
  • Receptors, Opioid, kappa / physiology*
  • Sex Characteristics
  • Sex Factors
  • Time Factors
  • alpha-MSH / metabolism*

Substances

  • Pyrrolidines
  • Receptors, Opioid, kappa
  • norbinaltorphimine
  • alpha-MSH
  • Naltrexone
  • 3,4-Dichloro-N-methyl-N-(2-(1-pyrrolidinyl)-cyclohexyl)-benzeneacetamide, (trans)-Isomer
  • Prolactin