T cell responses affected by aminopeptidase N (CD13)-mediated trimming of major histocompatibility complex class II-bound peptides

J Exp Med. 1996 Jul 1;184(1):183-9. doi: 10.1084/jem.184.1.183.

Abstract

Endocytosed protein antigens are believed to be fragmented in what appears to be a balance between proteolysis and MHC-mediated epitope protection, and the resulting peptide-MHC complexes are transported to the surface of the antigen-presenting cells (APC) and presented to T cells. The events that lead to antigenic peptide generation and the compartments where antigen processing takes place remains somewhat enigmatic. The importance of intracellular antigen processing has been well established; however, it is unclear whether additional processing occurs at the APC surface. To follow antigen processing, we have identified a pair of T cell hybridomas that recognize a long vs. a short version of the same epitope. We have used prefixed APC and various protease inhibitors to demonstrate that the APC surface has a considerable potential for antigen processing. Specific antibodies further identified the exopeptidase Aminopeptidase N (APN, CD13) as one of the enzymes involved in the observed cell-surface antigen processing. The NH2-terminal end of the longer peptide could, even while bound to major histocompatibility complex (MHC) class II molecules, be digested by APN with dramatic consequences for T cell antigen recognition. This could be demonstrated both in cell-free systems using purified reagents and in cellular systems. Thus, MHC class II and APN may act in concert to generate the final T cell epitopes.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Sequence
  • Animals
  • Antigen-Presenting Cells / enzymology*
  • CD13 Antigens / metabolism*
  • Histocompatibility Antigens Class II / metabolism*
  • Hybridomas
  • Lymphocyte Activation
  • Mice
  • Molecular Sequence Data
  • Muramidase / chemistry
  • Muramidase / immunology
  • Peptides / immunology*
  • Peptides / metabolism
  • T-Lymphocytes / immunology*
  • Tumor Cells, Cultured

Substances

  • Histocompatibility Antigens Class II
  • Peptides
  • Muramidase
  • CD13 Antigens