A specific beta 3-adrenoceptor agonist induces increased pancreatic islet blood flow and insulin secretion in rats

Eur J Pharmacol. 1996 Mar 18;298(3):287-92. doi: 10.1016/0014-2999(95)00802-0.

Abstract

In order to study the role of beta 3-adrenoceptor stimulation on insulin secretion in rats, plasma insulin level and islet blood flow were measured during treatment with CL 316243 which is chemically named disodium (R,R)-5-[2-[[2,3-(3-chlorophenyl)-2-hydroxyethyl]-amino]propyl]-1, 3-benzodioxole-2,2-dicarboxylate, a specific beta 3-adrenoceptor agonist. CL 316243 induced a marked increase in both islet blood flow and plasma insulin concentration without changes in whole pancreatic blood flow. This increase was totally prevented when the rats were pretreated with bupranolol, a beta 1, beta 2, beta 3-adrenoceptor antagonist, but not with nadolol, a beta 1, beta 2-adrenoceptor antagonist. We conclude that beta 3-adrenoceptor stimulation provokes a marked vasodilatation of microvessels in the islets of Langerhans, which in turn could contribute to the increase in insulin secretion.

MeSH terms

  • Adrenergic beta-Agonists / pharmacology*
  • Adrenergic beta-Antagonists / pharmacology
  • Animals
  • Bupranolol / pharmacology
  • Dioxoles / pharmacology*
  • Female
  • Insulin / blood
  • Insulin / metabolism*
  • Insulin Secretion
  • Islets of Langerhans / blood supply
  • Islets of Langerhans / drug effects*
  • Islets of Langerhans / metabolism
  • Nadolol / pharmacology
  • Rats
  • Rats, Wistar
  • Receptors, Adrenergic, beta / drug effects*
  • Receptors, Adrenergic, beta-3
  • Regional Blood Flow / drug effects

Substances

  • Adrenergic beta-Agonists
  • Adrenergic beta-Antagonists
  • Dioxoles
  • Insulin
  • Receptors, Adrenergic, beta
  • Receptors, Adrenergic, beta-3
  • disodium (R,R)-5-(2-((2-(3-chlorophenyl)-2-hydroxyethyl)-amino)propyl)-1,3-benzodioxole-2,3-dicarboxylate
  • Nadolol
  • Bupranolol