The majority of 22 Dutch high-risk breast cancer families are due to either BRCA1 or BRCA2

Eur J Hum Genet. 1996;4(4):225-30. doi: 10.1159/000472203.

Abstract

We have analyzed, by a combination of mutation and linkage analysis, the genetic basis of 22 breast cancer families in which at least 4 cases of either breast cancer diagnosed under the age of 60 or ovarian cancer had occurred. Chain-terminating mutations in BRCA1 were evidenced in 6 families, and posterior probabilities of > 0.90 of being linked to BRCA1 in 3. The breast versus ovarian cancer ratio in these 9 families was approximately 2:1. Among the remaining 13 families, significant linkage to markers flanking BRCA2 was established in the admixture test with a maximum multipoint lod score of 3.38, but there was no statistical evidence for genetic heterogeneity. The breast:ovarian cancer ratio in these families was 7:1, suggesting BRCA2 confers a much lower risk for ovarian cancer than does BRCA1. These results suggest that BRCA2 will explain a significant proportion of hereditary breast cancer in the Netherlands, and, together with BRCA1, account for the majority of all high-risk families.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • BRCA2 Protein
  • Breast Neoplasms / epidemiology
  • Breast Neoplasms / genetics*
  • Female
  • Genes, BRCA1*
  • Genetic Markers
  • Humans
  • Lod Score
  • Models, Genetic
  • Mutation
  • Neoplasm Proteins / genetics*
  • Netherlands / epidemiology
  • Ovarian Neoplasms / epidemiology
  • Ovarian Neoplasms / genetics*
  • Risk Factors
  • Sequence Analysis, DNA
  • Transcription Factors / genetics*

Substances

  • BRCA2 Protein
  • Genetic Markers
  • Neoplasm Proteins
  • Transcription Factors