Induction of tumor necrosis factor by a camptothecin derivative, irinotecan, in mice and human mononuclear cells

Anticancer Res. 1996 Sep-Oct;16(5A):2507-11.

Abstract

Irinotecan (CPT-11) has been reported to be cytotoxic to tumor cells through its inhibitory activity on type I DNA topoisomerase. CPT-11 has also been shown to have several unique biological activities apart from direct cytotoxicity. We investigated the ability of CPT-11 to induce tumor necrosis factor (TNF) production. Human peripheral blood mononuclear cells (MNCs) were incubated with LPS, CPT-11, or with vinblastin sulfate as a control. The priming effect of CPT-11 on endogenous production of TNF was examined by injecting the drug intravenously into mice, followed 3 hours by the injection of OK432. At a dose of 200-400 micrograms/kg, CPT-11 showed a significant priming effect. A significant amount of TNF was released when MNCs were incubated with 100-300 microM of CPT-11 for more than 4 hours, but not with vinblastin sulfate, indicating a triggering effect of TNF production on MNCs in vitro. These effects may be advantageous in cancer therapy.

MeSH terms

  • Animals
  • Antineoplastic Agents, Phytogenic / blood
  • Antineoplastic Agents, Phytogenic / pharmacology*
  • Camptothecin / analogs & derivatives*
  • Camptothecin / blood
  • Camptothecin / pharmacology
  • Enzyme-Linked Immunosorbent Assay
  • Humans
  • Irinotecan
  • Male
  • Mice
  • Mice, Inbred C3H
  • Monocytes / drug effects*
  • Monocytes / metabolism
  • Time Factors
  • Tumor Necrosis Factor-alpha / biosynthesis*

Substances

  • Antineoplastic Agents, Phytogenic
  • Tumor Necrosis Factor-alpha
  • Irinotecan
  • Camptothecin